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This leads to increased secretion of corticotropin-releasing factor (CRF) in the hypothalamus and causes increased levels of glucocorticoids both in the brain and peripherally, being translated into increased mitochondrial activity ( In the mitochondria, glucocorticoids form a complex with the antiapoptotic protein Bcl-2 to inhibit the formation of Bax-containing pores on the mitochondrial outer membrane

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