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[2] Other GLP-1s have similar profiles

Since the mutagenesis of M3 disrupted 1 putative WD40 out of 2, we also mutagenized this second site (M5) and both M3 and M5 motifs and found a reduction in WIPI2 as well as ATG16L1 binding and the absence of BCAS3 interaction (Figure 7C)

Other indications which physicians said could be the future of GLP-a research include nonalcoholic fatty liver disease (37 percent), osteoarthritis (30 percent), and chemical dependency (30 percent)

May 28 2013;13:89