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Our study has limitations such as small sample size and evaluation of only acute effect, lack of assessment of liraglutide blood concentration, and recruitment of patients with only two stages of chronic kidney disease

38.7%) and suicidal ideation (n = 73

JAMA 282:15681575 Clemmensen C, Chabenne J, Finan B et al (2014) GLP-1/glucagon coagonism restores leptin responsiveness in obese mice chronically maintained on an obesogenic diet

Research indicates that excessively hyperplastic and dysfunctional EAT exerts adverse cardiac effects primarily through two pathways: first, by serving as a source of pro-inflammatory mediators, and second, through direct cardiac mechanical effects, ultimately promoting cardiovascular disease (82)