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Biochem J (1906) 1(1):2838
Similar content being viewed by others Introduction SARS-CoV-2 contributes to sickness and death in COVID-19 patients partly by inducing a hyper-proinflammatory state in the host airway 1,2
Some individuals inherit a genetic characteristic known as the hungry gut phenotype, resulting in reduced levels of satiety after eating and faster gastric emptying compared to the general population.1 These individuals tend to respond more favorably when treated with glucagon-like peptide 1 (GLP-1) agonists and experience higher levels of weight loss with semaglutide, a GLP-1 agonist
Proposed exploratory objectives including functional and phenotypic characterization of SARS-CoV-2-specific immune cells, cytokine synthesis, cellular proliferation, and mucosal antibody concentrations
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