It has been revealed that prolonged exposure to stress triggers biological mechanisms, in particular those related to the regulation of the inflammatory cascade, which plays a key role in the pathogenesis of many diseases
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This altered environment exerts selective pressure, reshaping the microbial community by reducing beneficial commensal bacteria such as Bifidobacterium and Lactobacillus and promoting opportunistic pathogens, especially within the phylum Proteobacteria (78, 79)
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