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Pathways affecting lipid metabolism and cell junctions, such as mitogen-activated protein kinase (MAPK) signaling, peroxisome proliferator-activated receptor (PPAR) signaling, tight junction, extracellular matrix (ECM)receptor interactions, focal adhesion, and regulation of the actin cytoskeleton, were suggested to potentially contribute to IMF deposition [29]

Future work should directly assess redox shifts in brain tissue or cerebrospinal fluid to confirm central effects

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