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Based on these findings, it was recognized for the first time that impairment of antioxidant defenses (vitamin E-deficiency) and enhanced levels of PUFAs (substrates for LPO) did not just amplify the injury mechanism but switched cell death signaling typically centered around mitochondrial dysfunction (in mice on normal diets) to one dominated by acute LPO [70]

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Draw up your prescribed dose into the injection syringe

923 However, supplementation with exogenous PUFA to balance the PUFA/MUFA ratio increases the sensitivity of resistant tumors to ferroptosis