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Studies intended to probe the telomerase claim should include appropriate controls and, ideally, independent readouts of telomerase activity and telomere length rather than a single assay

This measurable deficit provides objective, biochemical proof of the mitochondrial dysfunction that patients experience, validating their symptoms and highlighting the carnitine shuttle as a primary target for therapeutic intervention

Conte center, MH-094268, NIH MH 084018, MH-105660, and MH-107730, as well as foundation supports from the Stanley and S/R to A.S.

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