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These data suggest the presence of two unique and very distinct neuronal circuitries within the AP and highlight the hypothesis of a local inhibitory network within the caudal hindbrain that could be exploited via GIPR activation to reduce hindbrain GLP-1Rmediated emesis and nausea (Fig

Diabetes 2000;49:17001708 [DOI] [PubMed] [Google Scholar] 102

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Augmenting NO synthesis through exogenous substances may improve skeletal muscle function and performance through improved blood flow, contractility, and mitochondrial respiration (Stamler and Meissner 2001)