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doi: 10.1177/0300060518756540

Proposed Mechanisms Preventing GABA receptor downregulation Counteracting compensatory NMDA glutamate receptor sensitization Stabilizing benzodiazepine receptor configuration (preventing shift to inverse agonist state) Upregulating GABAergic neurotransmission through pathways distinct from benzodiazepine action itself This tolerance prevention has profound clinical implications

While chronically low levels of glutathione can lead to cell damage and chronic disease, increased levels might prevent illnesses and upping production is often the treatment for many chronic diseases

This pressure gradient is highly regulated by a dual feedback system, which can be manipulated pharmacologically to upregulate water transport through the lens and subsequently increase the delivery of antioxidants to the core region of the lens to prevent age-related nuclear cataracts