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Our findings further indicate that TGF- and oxidative stress, but not TNF-, contribute to upregulation of GPX3 in lung fibrosis in a cell-type-specific manner

Approved ASO drugs target exon 45 (casimersen), 264,496 exon 51 (eteplirsen), 497,498 and exon 53 (golodirsen and viltolarsen) 80,263 to induce specific exon skipping in the dystrophin gene, thus delaying disease progression

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