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This chronic inflammation, driven by excessive NO signaling, may lead to tissue damage and further facilitate polyp progression, positioning these proteins as promising biomarkers for diagnosis
Once inside the cell, the complex interacts with intracellular copper-responsive pathways which is what enables the downstream gene-modulation effects to translate from topical application into measurable dermal changes
The disruptive nature of enterotoxins on TJ complexes can also lead to the movement of basolateral proteins to apical sites on epithelial cells where they may be utilised by pathogens or their toxins as alternative receptors facilitating infection (Muza-Moons et al
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