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In vitro experiments further confirmed that Rg3 treatment increased ULK1 and FUNDC1 expression in oxygen-glucose deprivation/reperfusion (OGD/R)-treated rat cardiomyocytes, triggered the phosphorylation of FUNDC1 at the Ser17 site via ULK1 to activate mitophagy, and facilitated FUNDC1-LC3 interactions, which resulted in the maintenance of mitochondrial homeostasis and energy metabolism and ameliorated HF (Wang X

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Immunofluorescence of Kim1 and SMA also showed that the degree of tubular injury and interstitial fibrosis was similar in both groups (Figure 4G)

Health Savings Account (HSA) or Flexible Spending Account (FSA) funds may be eligible for physician-supervised treatments, though individual plan terms vary