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ROS produced by acute pancreatitis stimulate NF-B, leading to excessive release of cytokines such as interleukin-1 (IL-1), interleukin-2 (IL-2), interleukin-18 (IL-18), interleukin-6 (IL-6), and TNF- [81]

This slow, controlled absorption supports consistent peptide delivery to the bloodstream, promoting more stable and sustained effects compared to injections or oral forms

Altogether, this contributes to neuroinflammation, oxidative damage, and neuronal death, all of which are observed in PD (La Vitola et al., 2024

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