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Key pathophysiological changes in acute lung injury caused by sepsis include infiltration of inflammatory cells, release of inflammatory mediators, thrombus formation in pulmonary capillaries, interstitial pulmonary edema and pulmonary fibrosis, with clinical manifestations primarily being progressive dyspnea and persistent hypoxemia secondary to sepsis (99)

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a , Photomicrograph of coronal NTS section from PPG-Cre:tdRFP mouse transduced with DIO-TVA-mCherry + DIO-RabiesG, and subsequently with rabies virus-G-GFP (RABV)

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