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Increased clearance of IgG in mice that lack beta(2)-microglobulin: possible protective role of FcRn
As the essential eNOS cofactor tetrahydrobiopterin (BH 4 ) is susceptible for oxidation, a landmark study by David Harrison's group 62 was able to proof the concept that superoxide produced by the Nox may indeed trigger eNOS uncoupling in the experimental animal model of DOCA-salt hypertension
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Degradation of integrin IIb appears to involve retro-translocation from the endoplasmic reticulum into the cytoplasm, ubiquitination, and proteolysis by the megakaryocyte proteasome
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