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Porphyromonas gingivalis and its LPS can translocate from the oral cavity to the gut, causing gut dysbiosis 111 , which is linked to increased hepatic expression of TLR2, TNF-, and IL-17, thereby promoting liver inflammation 112

Considering the paramount importance of oxLDL in the pathogenesis of atherosclerosis, it is reasonable to evaluate the role of antioxidants in the treatment of the disease as adjuvant strategies to lipid-lowering or anti-inflammatory therapies designed to reduce the risk of cardiovascular disease (230)

For instance, in the Dutch population, ApoE4 allele has been associated with an increased risk of FTLD (Stevens et al., 1997)