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Interestingly, RSL3-induced ferroptosis could be inhibited by higher expression levels of mitochondrial ferritin under hypoxic conditions [60]

SQSTM1/p62 and PPARGC1A/PGC-1alpha at the interface of autophagy and vascular senescence

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The growing pharmaceutical interest in this compound stems from its simultaneous targeting of multiple pathological processes, including oxidative stress, aberrant cellular proliferation, and inflammatory cascades, making it particularly valuable for complex disease conditions such as cancer, cardiovascular diseases, and cerebrovascular disorders including cerebral ischemia [2]