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Notably, sleep-deprived mice treated with GHK had lower inflammation compared to both sleep-deprived and control mice in the CA3 region of the hippocampus

Normally this would, in turn, lead to increased glutathione through glutathione synthetase, but it is thought that glutathione synthetase may become rate-limiting or may even be inhibited by acetaminophen or a product of its metabolism [9]

The review highlights FOXO4-DRI's unique mechanism as a peptide-based senolytic with high selectivity for senescent cells, while noting challenges for clinical translation including peptide delivery and stability

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