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Increasing both levels of ROS and reactive nitrogen species (RNS) through the destruction of cellular structures, such as DNA, proteins, and lipids, can increase the risk of migraine

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362 Considering the expression of the GLP-1R in thyroid C cells and the ability of GLP-1 to stimulate calcitonin secretion through a cAMP-mediated mechanism in vitro, it is plausible that calcitonin plays a role in the alterations in bone metabolism observed in GLP-1R-treated animals

non-empirical publications such as systematic reviews, meta-analyses, editorials, and opinion pieces