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In light of its role in the bi-directional transfer of information between the periphery and the brain, the PNS is well positioned to control energy metabolism, not only by modulating glycaemia via regulation of sympathetic outflow to the skeletal muscle 32 , but also by promoting GIP-induced vasodilation in the mesenteric vasculature, including the adipose tissue 33,34

Moreover, studies using mouse genetics have identified the importance of endothelial cell DPP-4 in the physiological control of glucose homeostasis and as a key cellular target for the pharmacological glucoregulatory response to DPP-4 inhibition (20)