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The accumulation of autophagosomes early after TBI reflects a deleterious effect in which impaired lysosomal function and autophagosome clearance lead to the accumulation of ubiquitinated proteins and protein aggregates, resulting in neuronal cell death

The D-retro-inverso design replaces all L-amino acids with D-amino acids and reverses the sequence a structural strategy that preserves the side-chain topology of the original FOXO4 binding region while making the peptide resistant to proteolytic degradation, dramatically extending plasma half-life compared to the natural L-form

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Gut-on-a-Chip microenvironment induces human intestinal cells to undergo villus differentiation