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These findings position the entero-gustatory-endocrine axis as a therapeutic target in metabolic disorders, where GLP-1 dysfunction disrupts taste signaling, perpetuating pathological feeding behaviors ( Figure 3 )

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In the 1 mg trial arm, liver fat fell 51% among participants with fatty liver, visceral fat fell 16%, triglycerides declined, and ketones rose as the liver used more fat. Modeled metabolic-health protocols place heavier, metabolically average profiles near 1 mg weekly