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To confirm that interface IV is essential for c-MET activation, we introduced Y673A mutation into HGF to disrupt this interface, and showed that HGF Y673 mutant has a great deficiency in binding to c-MET as well as promoting c-MET activation (Fig

It occurs naturally within the human body and has attracted considerable scientific interest due to its involvement in cellular communication, protein regulation and extracellular matrix activity

However, the same effect is observed when deleting or disrupting PRK1 ( CPAR2_800700 ), which encodes a putative protein kinase