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Eur Heart J 31:100111

It may cause damage and apoptosis of TECs through multiple mechanisms, including mitochondrial dysfunction, oxidative stress, regulation of HIF-1, and alteration of extracellular matrix ( 3.4 Inflammation Inflammation plays a crucial role in the pathophysiology of DKD

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In 2005, Peled and colleagues claimed that copper-free GHK maintained the clonogenic potential of stem cells while GHK-Cu increased cell copper by 2,162 % above the control value and caused stem cell differentiation [ 41 ]