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In addition, based on the activation of mitochondrial -FAO metabolic pathways to find potentially protective agents of VPA induced abnormal liver function

With a fatty-acyl moiety on the GLP-1 peptide backbone, it has an increased half-life as a result of DPP-4 resistance and non-covalent binding to serum albumin

The interaction between iron regulatory elements ( IREs ) and iron regulatory proteins ( IRPs ) ensures that iron is appropriately utilized and stored, further connecting heme metabolism with iron recycling [21, 22]

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