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Insulin resistance, hyperinsulinemia, dyslipidaemia, and obesity precede the progression to T2D in 7585 % of patients [65], and the presence of MetS increases up to fivefold the risk for T2D compared to individuals without MetS [66, 67]
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The peptide is chemically modified with an N-terminal tyrosine residue to enhance stability and contains a disulfide bond between cysteine residues
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