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Patients respond differently to the same interventions

[10] [11] Levels can be variable, even within species and some tissues can contain much more than others

Secondary iron overload Secondary iron overload is a phenomenon of excessive absorption and subsequent organ deposition, which is unrelated to a known genetic mutation: Chronic liver Diseases such as hepatitis C and B, alcohol liver disease, MASLD, and porphyria cutanea tarda downregulate hepcidin, resulting in increased iron absorption from the gastrointestinal tract

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