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In contrast to GLP-1, the incretin hormone glucose-dependent insulinotropic polypeptide (GIP) received little attention as a pharmacological target, because of conflicting observations that argue activation or inhibition of the GIP receptor (GIPR) provides beneficial effects on systemic metabolism

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Ces observations suggrent un potentiel anti-inflammatoire, mais il nexiste actuellement aucune validation de ces effets dans des essais cliniques humains

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