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In addition, exercise training can also prevent dysglycemia and impaired hepatic signaling after TBI (such as an increase in phosphorylated c-Jun NH2-terminal kinase, decrease in insulin receptor substrate, and phosphorylated AKT expression), and protect against hepatic inflammation (COX-2, iNOS, TNF-, and IL-6), and oxidative stress (decreases in non-protein sulfhydryl and glutathione, as well as increases in 2,7-dichlorofluorescein diacetate oxidation and protein carbonyl), demonstrating that exercise training alters the profile of oxidative-inflammatory status in the liver and protects against acute hyperglycemia and a cerebral inflammatory response after TBI [81]

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Longer for complicated cases

The gastric mucosa is a specialized epithelial layer that depends on continuous ECM remodeling and collagen synthesis for structural integrity