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Some studies involved trisomy 16 mouse models in order to get a basis before clinical trials on DS humans because these mice have increased oxidative stress and cerebral pathology similar to DS [51, 52, 53, 54]

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The cytosolic basal E GSH of 3D7 and Dd2 were estimated to be 314.23.1 mV and 313.93.4 mV, respectively, which is indicative of a highly reducing compartment