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Another case-control study reported that individuals in the highest quintile of riboflavin status, as measured by red blood cell glutathione reductase activity, had approximately one-half the occurrence of age-related cataract as those in the lowest quintile of riboflavin status, though the results were not statistically significant (34)

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Collectively, these findings support a bidirectional feedback loop: Inflammatory cues (such as IL-1 and oxidative stress) suppress DNMT expression and activity, eroding genomic methylation, while low folate or MTHFR-limited flux and the resulting homocysteine/SAM:SAH imbalance promote oxidative and NF-B signaling

Abbasnezhad A, Choghakhori R, Kashkooli S, Alipour M, Asbaghi O, Mohammadi R