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We note that the defect in RAD51 focus formation is not due to impaired DNA end resection as depletion of PDS5B and ectopic expression of PDS5B 17A did not significantly affect RPA focus formation after IR exposure (Fig

Results NOD mice manifest deficient responses to GLP-1 analogue exendin-4 GLP-1 and its analogues stimulate release of insulin from beta cells, which in turn allows uptake of glucose from the blood into tissues