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Hormonal treatment options include: Combined oral contraceptives : Continuous or cyclic regimens can reduce menstrual flow and suppress endometrial tissue activity Progestins : Available as oral medications, injections (depot medroxyprogesterone acetate), intrauterine devices (levonorgestrel-releasing IUD), or implants GnRH agonists : These medications induce a temporary menopausal state, effectively suppressing endometriosis but requiring add-back hormone therapy to minimize bone loss and vasomotor symptoms GnRH antagonists : FDA-approved oral options include elagolix (Orilissa) and relugolix/estradiol/norethindrone (Myfembree) with specific duration limits based on dosing and bone health considerations Aromatase inhibitors : Sometimes used in refractory cases, though not FDA-approved for this indication Bone mineral density monitoring is recommended for patients on long-term GnRH agonists or antagonists, particularly at higher doses or extended durations

Body fat begins declining measurably while lean mass remains stable or slowly increases (assuming adequate protein and training)

Where BPC-157 research focuses on blood vessel and growth-factor pathways, TB-500 research focuses on actin dynamics, the cytoskeletal machinery that lets cells change shape and migrate into a wound

95% CI, 95% confidence interval