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Glial cell activation in ME/CFS: implications for neuroinflammation Glial cells, particularly microglia and astrocytes, play a central role in the neuroinflammatory processes observed in ME/CFS
Gilbert syndrome It is an inherited disorder marked by slight, persistent unresolved hyperbilirubinemia without hemolysis or other signs of liver injury
The classic presentation of this condition is as follows: Intense immediate pain at injection Livedoid discoloration (mottled, dusky appearance) Rapid ecchymosis (bruising) Blistering and eventual skin necrosis This requires emergency specialist care
A first-in-human phase 0 clinical study of RNA interference-based spherical nucleic acids in patients with recurrent glioblastoma
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