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This blockade depletes the intracellular pool of lactate available for conversion into lactyl-CoA, thereby "starving" the epigenetic machinery required for lactylation and mitigating the aberrant proliferative response driven by metabolic stress [44, 67] Directly targeting the writer enzymes responsible for adding lactyl groups to proteins represents a precise epigenetic intervention strategy

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It also supports cardiovascular health by regulating blood pressure and reducing the risk of heart disease

doi:10.1093/jn/134.3.489