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Mitophagy usually induces an abnormal state during the occurrence and development of NDD, leading to excessive accumulation of dysfunctional mitochondria in neurons, triggering neuronal necrosis and OS, and aggravating neuronal damage ( In vivo experiments demonstrated that MORIN treatment significantly upregulated the expression of Parkin and LC3-II in the frontal cortex and hippocampus of Parkinsons disease (PD) mice, downregulated the expression of Mfn1, increased the expression of tyrosine hydroxylase (TH) protein, facilitated mitophagy, attenuated the loss of dopaminergic neurons, and ameliorated the behavioral deficits in PD mice (Wang ZY

2006 Apr 27;354(17):178795

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