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Free radicals generated by ultraviolet radiation, pollution, and other environmental stressors cause oxidative damage to cellular structures, including DNA, proteins, and lipids that maintain healthy skin function

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Specifically, in ischemic/high-ROS environments, BACH1 tends to be degraded and acts to suppress angiogenesis by inhibiting the Wnt/-catenin signaling pathway and recruiting HDAC1, among other mechanisms

Mice tolerated doses several times higher than the effective dose with no observable adverse effects