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However, the molecular mechanisms underlying the pathological aggregation of fat within the liver are not fully elucidated

357 Similarly, in HD and related polyQ disorders, expanded polyQ-containing proteins form insoluble aggregates that physically inhibit proteasome function, amplifying the proteotoxic burden

By delving into the regulatory processes of ferroptosissuch as iron ion metabolism, ROS generation, and the equilibrium of antioxidant defenseswe can gain clearer insights into its involvement in eye diseases