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Calabro FJ, Parr AC, Sydnor VJ, Hetherington H, Prasad KM, Ibrahim TS, et al

Activation of ferroptosis in diabetic patients increases the vulnerability of the heart after I/R

In AP, mitochondrial dysfunction, including ATP depletion, mtROS overproduction, and loss of m, promotes nuclear activation of stress-responsive transcriptional programs that enhance cytokine and chemokine expression and thereby contribute to pancreatic inflammation ( ATF4 is a key effector of the integrated stress response that connects mitochondrial stress, ER stress, and redox imbalance to nuclear transcriptional reprogramming