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NF-B not only induces canonical pro-inflammatory cytokines (TNF-, IL-6, pro-IL-1) but simultaneously up-regulates facilitative glucose transporter, metabolically locking macrophages, DCs and activated T cells into a glycolytic, NO-rich phenotype that fuels chronic mucosal inflammation (11, 110112)

Inhibition of the system xc GSHGPX4 axis can predispose cells to ferroptosis, while the primary initiation of ferroptosis typically occurs through the accumulation of toxic lipids, as discussed later in the section

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