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PRMT1 catalyzes the mono- and asymmetric dimethylation of arginine residues, thereby influencing protein function, stability, and subcellular localization [14, 15]
162 During ER stress, the UPR modulates FOXO1 through XBP1u: its loss stabilizes FOXO1 and promotes sustained autophagy
It is a prescription drug for its approved use
The distinct physical activity patterns observed in ME/CFS, characterized by reduced activity intensity but not increased sedentary behavior, further highlight the link between PEN and metabolic dysfunction, likely driven by issues with energy metabolism (Newton et al., 2011)
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