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Therefore, despite its hyperglycemic effects, glucagon triggers lipid catabolism, energy expenditure and reduces food intake

Once released, GLP-1 enters the bloodstream and attaches to specific GLP-1 receptors

We propose that chronic exposure to a particular suite of environmental and genetic factors elicits a trajectory of impaired mitochondrial biology that maximally impacts subsets of mitotypes abundant in the hippocampus, entorhinal cortex, and cerebral cortex, which eventually surfaces as the hallmark features of AD

The moment a patient hits a dose that causes troublesome nausea or loss of appetite beyond whats manageable, Nooms clinician-designed protocol holds at that dose longer, or even dials back to the prior dose until symptoms subside