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Under this condition, HIF-1 becomes stabilized and subsequently translocates to the nucleus, where it binds to hypoxia-responsive elements within target genes, thereby promoting metabolic programming, notably the upregulation of anaerobic glycolysis (37,38)

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Phase Ib/2 Study of Oral Decitabine/Cedazuridine (ASTX727) and Venetoclax in Combination with the Targeted Mutant IDH1 Inhibitor Ivosidenib or the Targeted Mutant IDH2 Inhibitor Enasidenib in IDH Mutated Acute Myeloid Leukemia