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They express more GH receptors than subcutaneous fat cells and are more responsive to lipolysis signals

Therefore, riboflavin deficiency will lead to the downregulation of GSH and oxidative stress, which is the predominant cause for reduced osteogenic capacities of BMSCs and enhanced osteoclastic potentials of osteoclast precursors [54, 55], finally leading to net bone loss

The benefit persisted at the 12-month follow-up