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Following an acquired brain injury, acute glutamate release induces excitotoxicity, neuronal injury, cell death, impairing the function and metabolism of surviving neurons and potentiating KP dysregulation [251, 252]

doi: 10.1155/2016/3758278 315 SantulliG

pylori infection, exploring different cell death signaling pathways and gaining deeper insights into their importance in the pathogenesis of the disease, and searching for biologically active substances that can inhibit or induce PCD may be crucial for future treatments of H