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Summary and prospects This article comprehensively outlines the pathogenic role of Th17/Treg imbalance in AR, and reviews the targeted treatment strategies in recent years, but also emphasizes the limitations of this field and the major obstacles to its clinical transformation

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FAK-paxillin system Studies at the molecular level suggest the interaction of BPC-157 with FAK (Focal Adhesion Kinase) and paxillin proteins involved in cell adhesion and signal transduction