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Repurposing GLP-1 Receptor Agonists for Parkinsons Disease: Current Evidence and Future Opportunities

Furthermore, the intestinal release of GLP-1 is stimulated by the gut microbiota, which use undigested food nutrients, fiber, and bile acids to produce metabolites and induce further secretion of GLP-1 from enteroendocrine L cells [24]

Rahat-Rozenbloom S, Fernandes J, Cheng J, Wolever TMS Acute increases in serum colonic short-chain fatty acids elicited by inulin do not increase GLP-1 or PYY responses but may reduce ghrelin in lean and overweight humans

Gastrointestinal Effects of GLP-1 Receptor Agonists: Mechanisms and Clinical Implications