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Rather than representing a purely cutaneous condition, PsO appears to be a systemic metabolicinflammatory disease in which insulin resistance, adipokine imbalance, and altered gutendocrine signaling contribute to immune dysregulation long before the onset of clinically overt joint involvement (7)

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Lixisenatide, another GLP-1 analogue, has also been reported to decrease atheroma plaque size and instability in animal models, by reprogramming macrophages towards M2 phenotype (10 g/kg/day) (30)

Conclusion This comprehensive review highlights the therapeutic potential of GLP-1 and its analogs in the management of metabolic disorders, particularly T2DM