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However, these therapeutic systems use a paracellular pathway or nonspecific passive transcellular pathway, resulting in a low peptide absorption rate and high variability in absorption

In smoke-induced pulmonary fibrosis models, tranilast treatment significantly reduced inflammatory cytokines (IL-1, TNF-, TGF-1), oxidative stress, and NF-B expression, while also inhibiting fibroblast proliferation in lung tissue [150]

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